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An Ancient Viral Protein May Play a Key Role in ALS Researchers find that UBQLN2 gene dysfunction causes a virus-like protein to accumulate in cells, which changes gene expression and may contribute to disease progression in amyotrophic lateral sclerosis (ALS). Read more here in The Scientist .

CU Boulder researchers have identified a surprising new player in the disease鈥攁n ancient, virus-like protein best known, paradoxically, for its essential role in enabling placental development. The findings were recently published in the journal eLife .